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Tubular handling of sodium and phosphate





Tubular handling of sodium and phosphate in non-ascitic liver

10/24/2014
06:34 | Author: Evan Martin

Phosphate transport
Tubular handling of sodium and phosphate in non-ascitic liver

Scand J Clin Lab Invest. 1987 May;47(3):247-51. Tubular handling of sodium and phosphate in non-ascitic liver cirrhosis. Caregaro L, Angeli P, Merlo A, Menon.

Fractional phosphate excretion was not impaired in our patients, and no correlation was found between phosphate excretion and proximal sodium reabsorption, as evaluated by clearance methods. In spite of a normal sodium balance during the pre-study period, non-ascitic cirrhotics showed a blunted proximal natriuretic response to maximal water load. Abnormalities in tubular handling of phosphate may account for the dissociation between proximal sodium reabsorption and phosphate excretion during hypotonic diuresis in these patients. This study demonstrates that an increased reabsorption of sodium in the proximal tubule is responsible for the impaired response to maximal water load in non-ascitic cirrhotics. Since phosphate excretion has been proposed as a proximal marker in liver cirrhosis, sodium reabsorption in the proximal tubule was compared with phosphate fractional excretion. The renal response to a maximal water load was evaluated in eight cirrhotic patients free of ascites and without previous evidence of ascites and in seven controls. Fractional sodium reabsorption in the proximal and diluting segment was estimated by clearance methods during hypotonic diuresis. In fact sodium excretion during hypotonic diuresis was reduced (p less than 0.05) and proximal sodium reabsorption increased (p less than 0.005) in cirrhotics.

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Tubular handling of sodium and phosphate in cirrhosis with salt

12/23/2014
04:08 | Author: Evan Martin

Sodium and phosphate formula
Tubular handling of sodium and phosphate in cirrhosis with salt

Tubular handling of sodium and phosphate were studied in 4 patients with cirrhosis and ascites. The control group consisted of 5 patients with cirrhosis without.

This observation suggests that sodium retention in these patients occurs beyond the proximal tubule. This interpretation is in accord with our previous observation, based on clearance data, that the proximal tubular reabsorption of sodium in cirrhosis may be normal even in the face of edema formation. The control group consisted of 5 patients with cirrhosis without sodium retention. The degree of phosphaturia was assumed to reflect proximal tubular reabsorption. Whereas fractional excretion of phosphate was comparable in both groups, fractional excretion of sodium was strikingly diminished in the patients with ascites. Tubular handling of sodium and phosphate were studied in 4 patients with cirrhosis and ascites.

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National Center for Biotechnology Information, U.S. National Library of Medicine 8600 Rockville Pike, Bethesda MD, 20894 USA.

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PDF Plus - Informa Healthcare

10/22/2014
02:38 | Author: Caitlin White

Phosphate transport
PDF Plus - Informa Healthcare

Tubular handling of sodium and phosphate in non-ascitic liver cirrhosis. Scand J. Clin Lab Invest 1987; 47: 247-251. The renal response to a maximal water load.

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Tubular handling of sodium and phosphate in non - ResearchGate

8/21/2014
12:04 | Author: Devin Garcia

Phosphate transport
Tubular handling of sodium and phosphate in non - ResearchGate

ABSTRACT The renal response to a maximal water load was evaluated in eight cirrhotic patients free of ascites and without previous evidence of ascites and in.

Fractional phosphate excretion was not impaired in our patients, and no correlation was found between phosphate excretion and proximal sodium reabsorption, as evaluated by clearance methods. Fractional sodium reabsorption in the proximal and diluting segment was estimated by clearance methods during hypotonic diuresis. In spite of a normal sodium balance during the pre-study period, non-ascitic cirrhotics showed a blunted proximal natriuretic response to maximal water load. Abnormalities in tubular handling of phosphate may account for the dissociation between proximal sodium reabsorption and phosphate excretion during hypotonic diuresis in these patients. In fact sodium excretion during hypotonic diuresis was reduced (p less than 0.05) and proximal sodium reabsorption increased (p less than 0.005) in cirrhotics. ABSTRACT The renal response to a maximal water load was evaluated in eight cirrhotic patients free of ascites and without previous evidence of ascites and in seven controls. Since phosphate excretion has been proposed as a proximal marker in liver cirrhosis, sodium reabsorption in the proximal tubule was compared with phosphate fractional excretion. This study demonstrates that an increased reabsorption of sodium in the proximal tubule is responsible for the impaired response to maximal water load in non-ascitic cirrhotics.

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Approach to Renal Tubular Disorders - ResearchGate

6/20/2014
02:10 | Author: Caitlin White

Phosphate transport
Approach to Renal Tubular Disorders - ResearchGate

Tubular function tests involve evaluation of functions of the proximal tubule (i.e., tubular handling of sodium, glucose, phosphate, calcium, bicarbonate and.

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